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An in vitro model yields ‘importin’ new insights into chronic traumatic encephalopathy: damaged astrocytes stop ‘thrombospondin’ to the injury
Author(s) -
Jaber Sausan M.,
Polster Brian M.
Publication year - 2017
Publication title -
journal of neurochemistry
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.75
H-Index - 229
eISSN - 1471-4159
pISSN - 0022-3042
DOI - 10.1111/jnc.13906
Subject(s) - chronic traumatic encephalopathy , in vitro , traumatic brain injury , neuroscience , thrombospondin , encephalopathy , medicine , biology , microbiology and biotechnology , poison control , psychiatry , biochemistry , injury prevention , concussion , medical emergency , metalloproteinase , matrix metalloproteinase
Read the highlighted article ‘Defective synthesis and release of astrocytic thrombospondin‐1 mediates the neuronal TDP‐43 proteinopathy, resulting in defects in neuronal integrity associated with chronic traumatic encephalopathy: in vitro studies’ on page 645.