An  in vitro  model yields ‘importin’ new insights into chronic traumatic encephalopathy: damaged astrocytes stop ‘thrombospondin’ to the injury | Zendy

Jaber Sausan M. | Zendy; Polster Brian M. | Zendy

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An in vitro model yields ‘importin’ new insights into chronic traumatic encephalopathy: damaged astrocytes stop ‘thrombospondin’ to the injury

Author(s) -

Jaber Sausan M.,

Polster Brian M.

Publication year - 2017

Publication title -

journal of neurochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.75

H-Index - 229

eISSN - 1471-4159

pISSN - 0022-3042

DOI - 10.1111/jnc.13906

Subject(s) - chronic traumatic encephalopathy , in vitro , traumatic brain injury , neuroscience , thrombospondin , encephalopathy , medicine , biology , microbiology and biotechnology , poison control , psychiatry , biochemistry , injury prevention , concussion , medical emergency , metalloproteinase , matrix metalloproteinase

Read the highlighted article ‘Defective synthesis and release of astrocytic thrombospondin‐1 mediates the neuronal TDP‐43 proteinopathy, resulting in defects in neuronal integrity associated with chronic traumatic encephalopathy: in vitro studies’ on page 645.

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