Premium
Effect of professional mechanical plaque removal on secondary prevention of periodontitis and the complications of gingival and periodontal preventive measures
Author(s) -
Sanz Mariano,
Bäumer Amelie,
Buduneli Nurcan,
Dommisch Henrik,
Farina Roberto,
Kon Eija,
Linden Gerard,
Meyle Joerg,
Preshaw Philip M.,
Quirynen Marc,
Roldan Silvia,
Sanchez Nerea,
Sculean Anton,
Slot Dagmar Else,
Trombelli Leonardo,
West Nicola,
Winkel Edwin
Publication year - 2015
Publication title -
journal of clinical periodontology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.456
H-Index - 151
eISSN - 1600-051X
pISSN - 0303-6979
DOI - 10.1111/jcpe.12367
Subject(s) - gingival recession , medicine , dentistry , dental plaque , dentine hypersensitivity , dentin hypersensitivity , periodontitis , oral hygiene , gingival margin , occlusion , chronic periodontitis , dentin , surgery
Background and Aims The scope of this working group was to review: (1) the effect of professional mechanical plaque removal ( PMPR ) on secondary prevention of periodontitis; (2) the occurrence of gingival recessions and non‐carious cervical lesions ( NCCL ) secondary to traumatic tooth brushing; (3) the management of hypersensitivity, through professionally and self administered agents and (4) the management of oral malodour, through mechanical and/or chemical agents. Results and Conclusions Patients undergoing supportive periodontal therapy including PMPR showed mean tooth loss rates of 0.15 ± 0.14 teeth/year for 5‐year follow‐up and 0.09 ± 0.08 teeth/year (corresponding to a mean number of teeth lost ranging between 1.1 and 1.3) for 12–14 year follow‐up. There is no direct evidence to confirm tooth brushing as the sole factor causing gingival recession or NCCL s. Similarly, there is no conclusive evidence from intervention studies regarding the impact of manual versus powered toothbrushes on development of gingival recession or NCCL s, or on the treatment of gingival recessions. Local and patient‐related factors can be highly relevant in the development and progression of these lesions. Two modes of action are used in the treatment of dentine hypersensitivity: dentine tubule occlusion and/or modification or blocking of pulpal nerve response. Dentifrices containing arginine, calcium sodium phosphosilicate, stannous fluoride and strontium have shown an effect on pain reduction. Similarly, professionally applied prophylaxis pastes containing arginine and calcium sodium phosphosilicate have shown efficacy. There is currently evidence from short‐term studies that tongue cleaning has an effect in reducing intra‐oral halitosis caused by tongue coating. Similarly, mouthrinses and dentifrices with active ingredients based on Chlorhexidine, Cetylpyridinium chloride and Zinc combinations have a significant beneficial effect.