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Transient renal tubulopathy in a Labrador retriever
Author(s) -
Jamieson P. M.,
Chandler M. L.
Publication year - 2001
Publication title -
journal of small animal practice
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.7
H-Index - 67
eISSN - 1748-5827
pISSN - 0022-4510
DOI - 10.1111/j.1748-5827.2001.tb06025.x
Subject(s) - medicine , tubulopathy , polyuria , polydipsia , labrador retriever , aminoaciduria , proteinuria , renal function , fanconi syndrome , cystinuria , endocrinology , urinary system , urology , metabolic acidosis , gastroenterology , urine , pathology , kidney , diabetes mellitus , biochemistry , cystine , enzyme , chemistry , cysteine
A four‐month‐old male Labrador retriever was presented for polyuria, polydipsia and persistent euglycaemic glucosuria. On referral, diagnostic tests demonstrated abnormal fractional excretions of electrolytes, increased urinary excretion of selected amino acids, mild renal tubular acidosis and mild proteinuria, indicating renal tubular dysfunction. Pyelonephritis was suspected and potentiated amoxycillin was administered. On reevaluation at six months of age, the dog was no longer polyuric or polydipsic and the metabolic abnormalities associated with the tubulopathy had resolved. Transient Fanconi's syndrome has not previously been reported in small animals. This report demonstrates the potential for recovery of function in cases presenting with renal tubulopathies.