Premium
Pathophysiology of the gastric dilation‐torsion complex in the dog *
Author(s) -
Wingfield W. E.,
Cornelius L. M.,
Deyoung D. W.
Publication year - 1974
Publication title -
journal of small animal practice
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.7
H-Index - 67
eISSN - 1748-5827
pISSN - 0022-4510
DOI - 10.1111/j.1748-5827.1974.tb05659.x
Subject(s) - medicine , pathophysiology , perfusion , cardiology , dilation (metric space) , hemorrhagic shock , shock (circulatory) , mathematics , combinatorics
The gastric dilation‐torsion complex in the dog involves a series of related pathophysiological changes which may culminate in death if therapy is not promptly initiated. This complex probably starts with gastric dilation which may or may not lead to gastric torsion. Gastric dilation interferes with blood flow through the caudal vena cava as well as the portal vein. Venous return to the heart is decreased, cardiac output decreases, and arterial hypotension ensues. With the onset of this hypotension, the animal is subjected to impairment of tissue perfusion, cellular catabolism, and decreased renal function. In addition, endotoxic shock and disseminated intravascular coagulation may occur. These two processes will likely lead to death of the animal.