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Investigation of the functional variant c.‐169T > C of the Fc receptor‐like 3 ( FCRL3 ) gene in alopecia areata
Author(s) -
Schäfer N.,
Blaumeiser B.,
Becker T.,
FreudenbergHua Y.,
Hanneken S.,
Eigelshoven S.,
Schmael C.,
Lambert J.,
De Weert J.,
Kruse R.,
Nöthen M. M.,
Betz R. C.
Publication year - 2006
Publication title -
international journal of immunogenetics
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.41
H-Index - 47
eISSN - 1744-313X
pISSN - 1744-3121
DOI - 10.1111/j.1744-313x.2006.00633.x
Subject(s) - alopecia areata , rheumatoid arthritis , immunology , autoimmune disease , medicine , gene , lupus erythematosus , genetics , biology , antibody
A functional variant in the Fc receptor‐like 3 ( FCRL3 ) gene has been implicated in susceptibility to autoimmune diseases such as rheumatoid arthritis, systemic lupus erythematosus and autoimmune thyroid disease. Investigating a large case‐control sample of patients with alopecia areata (AA), we found no evidence for the involvement of FCRL3 in susceptibility to AA.