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Cardiac electromechanical delay is increased during recovery from 40 km cycling but is not mediated by exercise intensity
Author(s) -
ChanDewar F.,
Gregson W.,
Whyte G.,
King J.,
Gaze D.,
CarranzaGarcía L. E.,
LegazArrese A.,
George K.
Publication year - 2013
Publication title -
scandinavian journal of medicine and science in sports
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.575
H-Index - 115
eISSN - 1600-0838
pISSN - 0905-7188
DOI - 10.1111/j.1600-0838.2011.01376.x
Subject(s) - cycling , cardiology , medicine , intensity (physics) , troponin i , diastolic function , qrs complex , exercise intensity , diastole , heart rate , blood pressure , physics , myocardial infarction , archaeology , quantum mechanics , history
Cardiac electrical‐mechanical delay ( cEMD ), left ventricular ( LV ) function, and cardiac troponin I ( cTnI ) were assessed after 40 km cycle time trials completed at high ( HIGH ) and moderate ( MOD ) intensities in 12 cyclists. Echocardiograms and blood samples were collected before, 10, and 60 min after cycling. cEMD as assessed by time from QRS onset to peak systolic ( S ’) tissue velocity was lengthened after both bouts of cycling but was not mediated by cycling intensity ( HIGH : 174 ± 52 vs 198 ± 26 ms; MOD : 151 ± 40 vs 178 ± 52 ms, P < 0.05). Global LV systolic function was unaltered by exercise. cEMD from QRS to peak early ( E ’) diastolic tissue velocity was also increased post‐exercise ( HIGH : 524 ± 95 vs 664 ± 68 ms; MOD : 495 ± 62 vs 604 ± 91 ms, P < 0.05). Indices of LV diastolic function was reduced after cycling but were not mediated by exercise intensity. cTnI was elevated in two participants after HIGH trial (0.06 ug/ L ; 0.04 ug/ L ) and one participant after MOD trial (0.02 ug/ L ). While cEMD is lengthened and LV diastolic function was reduced post‐cycling, altering time‐trial intensity had little impact upon cEMD , LV function, and cTnI release.