Premium
Exercise‐induced arterial hypoxemia is not different during cycling and running in triathletes
Author(s) -
Laursen Paul B.,
Rhodes Edward C.,
Langill Robert H.,
Taunton Jack E.,
McKenzie Donald C.
Publication year - 2005
Publication title -
scandinavian journal of medicine and science in sports
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.575
H-Index - 115
eISSN - 1600-0838
pISSN - 0905-7188
DOI - 10.1111/j.1600-0838.2004.00391.x
Subject(s) - cycling , hypoxemia , medicine , respiratory exchange ratio , treadmill , cardiology , respiratory minute volume , vo2 max , respiratory system , physical therapy , heart rate , blood pressure , archaeology , history
This study examined the effect of running and cycling on exercise‐induced arterial hypoxemia (EIAH) in individuals well trained in each modality. Thirteen male triathletes (x̄±SD: age=36±5 years, mass=69±8 kg, body fat=12±1%) performed progressive exercise to exhaustion during cycle ergometry and treadmill running. Gas exchange was determined, while oxyhemoglobin saturation (SaO 2 ) was measured with an ear oximeter. At maximal exercise, the respiratory exchange ratio (1.15±0.06 vs. 1.10±0.05) and the ventilatory equivalent for oxygen uptake (37.6±3.8 vs. 34.2±2.7) were greater during cycling vs. running ( P <0.05). However, there were no differences at maximal exercise in oxygen uptake (64.4±3.2 vs. 67.0±4.6 mL kg −1 min −1 ), SaO 2 (93.4±2.8% vs. 92.6±2.2%), or the ventilatory equivalent for carbon dioxide (V̇ E /V̇CO 2 ; 33.1±3.1 vs. 31.0±3.1), during cycling vs. running, respectively. During submaximal exercise, the V̇ E /V̇CO 2 was less for cycling (26.0±1.0) compared with running (29.1±0.4; P <0.05), but this had no apparent effect on the SaO 2 response. In conclusion, EIAH was not significantly different during cycling and running in athletes who were well trained in both exercise modalities.