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Effect of fluoridated carbamide peroxide gels on enamel microtensile bond strength
Author(s) -
Chuang ShuFen,
Chen HuiPing,
Chang ChihHan,
Liu JiaKuang
Publication year - 2009
Publication title -
european journal of oral sciences
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.802
H-Index - 93
eISSN - 1600-0722
pISSN - 0909-8836
DOI - 10.1111/j.1600-0722.2009.00645.x
Subject(s) - enamel paint , demineralization , bond strength , fluoride , dentistry , carbamide peroxide , remineralisation , chemistry , adhesive , materials science , composite material , medicine , inorganic chemistry , layer (electronics)
The aim of this study was to examine the resin bond strength on enamel treated with different fluoridated bleaching agents. Forty‐eight bovine incisors were divided into four groups to receive bleaching treatments, over a 14‐d period, as follows: no treatment; 10% carbamide peroxide (CP) bleaching; 10% CP containing 0.11% fluoride; and 10% CP containing 0.37% fluoride. Immediately, and 7 and 14 d after bleaching, the enamel surfaces were respectively bonded with composite and sectioned to create resin–enamel beams. These beams were subjected to the microtensile bond strength (μTBS) test, then assessed for failure mode under scanning electron microscopy. The results showed that the 0.37% fluoridated group demonstrated a μTBS equivalent to that of the unbleached group at all stages. Non‐fluoridated and 0.11% fluoridated groups showed a weaker μTBS after bleaching but regained the bond strength after 14 or 7 d of storage, respectively. In the non‐fluoridated group, adhesive failure was the predominant fracture pattern that comprised the enamel prism demineralization change and widely dispersed voids on the resin–enamel interfaces. No evident enamel erosion and fewer microporosities were found in the 0.37% fluoridated group. Accordingly, treatment with 0.37% fluoridated CP maintained the μTBS as effectively as the unbleached enamel. Additional fluoride in the bleaching agents may facilitate subsequent restorative treatment by inhibiting enamel demineralization.