Premium
Is there a relationship between juvenile idiopathic arthritis and periodontitis?
Author(s) -
Reichert Stefan,
Machulla Helmut K. G.,
Fuchs Corinna,
John Vera,
Schaller HansGünter,
Stein Jamal
Publication year - 2006
Publication title -
journal of clinical periodontology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.456
H-Index - 151
eISSN - 1600-051X
pISSN - 0303-6979
DOI - 10.1111/j.1600-051x.2006.00909.x
Subject(s) - medicine , arthritis , clinical attachment loss , gastroenterology , periodontitis , juvenile , c reactive protein , bleeding on probing , dentistry , inflammation , genetics , biology
Aim: The aim was to compare the prevalence of periodontal conditions in patients with juvenile idiopathic arthritis (JIA) ( n =78, age 14.4 years) with those revealed in a healthy control group ( n =75, age 15.5 years). Material and Methods: In both groups, the approximal plaque index (API), the modified sulcular bleeding index (SBI), and the clinical attachment loss (CAL) were determined. Laboratory parameters for JIA activity included the capsule‐reactive protein (CRP) and the immunoglobulins A, G, M. Results: JIA patients had a significantly higher API (64.6% versus 49.9%, p =0.004) and slightly higher mean percentages of sites with CAL>3.5 mm (0.58% versus 0.22%, p =0.041). There was no significant difference in the prevalence of patients and controls who had sites with CAL >3.5 mm (25.6% versus 17.3%, p =0.212). The mean CAL was slightly greater (0.2 mm; p =0.030) in patients with CRP5.0 mg/l compared with patients with CRP<5.0 mg/l. Patients who took non‐steroidal anti‐inflammatory drugs (NSAIDs) had a significantly decreased SBI (26.2% versus 51.1%, p =0.019). Conclusion: After adjustment for microbial plaque, JIA is not a risk factor for periodontitis.