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Transient Crossed Cerebellar Diaschisis Following Thalamic Hemorrhage
Author(s) -
Takasawa Masashi,
Hashikawa Kazuo,
Ohtsuki Toshiho,
Imaizumi Masao,
Oku Naohiko,
Kitagawa Kazuo,
Hori Masatsugu,
Matsumoto Masayasu
Publication year - 2001
Publication title -
journal of neuroimaging
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.822
H-Index - 64
eISSN - 1552-6569
pISSN - 1051-2284
DOI - 10.1111/j.1552-6569.2001.tb00076.x
Subject(s) - internal capsule , medicine , diaschisis , thalamus , lesion , hematoma , perfusion , edema , cerebellum , radiology , nuclear medicine , magnetic resonance imaging , pathology , white matter , surgery
This report concerns a 65‐year‐old right‐handed woman with cerebral hemorrhage who presented with mild right‐sided hemiparesis. Computed tomography (CT) revealed hematoma in the left thalamus and compression of the posterior limb of the internal capsule by a brain edema surrounding the lesion. 99m Tc‐hexamethylpropyleneamine oxime (HMPAO) single photon emission computed tomography (SPECT) images obtained 4 days after onset showed hypoperfusion in the left thalamus containing a hematoma as well as contralateral cerebellar hypoperfusion to the supratentorial lesion, which is well recognized as crossed cerebellar diaschisis (CCD) after stroke. CT 14 days after the onset revealed reduction of the brain edema of the posterior limb of the internal capsule accompanied by gradual neurological improvement. SPECT obtained 14 and 28 days later showed that CCD had disappeared. In this case report, the authors discuss the disappearance of CCD due to transient edematous compression of the internal capsule following thalamic hemorrhage on serial 99m Tc‐HMPAO SPECT scans. CCD was possibly caused by the lesion confined to the posterior limb of the internal capsule, which anatomically constitutes the cerebropontocerebellar pathway.