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Catheter Ablation for an Incessant Form of Antiarrhythmic Drug‐Resistant Ventricular Fibrillation After Acute Coronary Syndrome
Author(s) -
ENJOJI YOSHIHISA,
MIZOBUCHI MASAHIRO,
SHIBATA KENSAKU,
YOKOUCHI ITARU,
FUNATSU ATSUSHI,
KANBAYASHI DAISUKE,
KOBAYASHI TOMOKO,
NAKAMURA SHIGERU
Publication year - 2006
Publication title -
pacing and clinical electrophysiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.686
H-Index - 101
eISSN - 1540-8159
pISSN - 0147-8389
DOI - 10.1111/j.1540-8159.2006.00286.x
Subject(s) - medicine , ventricular fibrillation , cardiology , ventricular tachycardia , catheter ablation , ablation , acute coronary syndrome , anesthesia , myocardial infarction
A 77‐year‐old man was admitted with an acute coronary syndrome (ACS), severe heart failure (HF), and repeated ventricular fibrillation (VF) episodes. A single premature ventricular complex (PVC) induced ventricular tachycardia (VT), which degenerated to VF reproducibly. This PVC was eliminated by catheter ablation at the left ventricular posteroseptal region where double Purkinje potentials preceding the ventricular wave had been recorded. The electrical storm disappeared, and programmable stimulation failed to induce any tachyarrhythmias after the ablation. A Purkinje fiber network‐related PVC served as a trigger and as a substrate for VT and VF in a case of ACS with HF.