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Characteristics and Clinical Effects of Myopotential Signals in a Unipolar DDD Pacemaker Population
Author(s) -
ZIMMERN SAMUEL H.,
CLARK MARTHA F.,
AUSTIN W. KENNETH,
FEDOR JOHN M.,
GALLAGHER JOHN J.,
SVENSON ROBERT H.,
DUNCAN JAMES L.
Publication year - 1986
Publication title -
pacing and clinical electrophysiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.686
H-Index - 101
eISSN - 1540-8159
pISSN - 0147-8389
DOI - 10.1111/j.1540-8159.1986.tb06664.x
Subject(s) - medicine , cardiology
Myopotential signals were recorded from atrial and ventricular leads during isometric exercise in 25 patients who had chronically implanted dual chamber pacemakers using the electrogram telemetry capability of the pacemakers. Average electrogram amplitude on the atrial channel was 0.92 mV (range 0.3 to 1.9) and on the ventricular channel was 0.98 mV (range 0.3 to 2.2); the difference was not significant. There was a strong correlation (R = 0.82) between the amplitude of myopotentials on the atrial and ventricular leads for individual patients. Myopotential sensing caused ventricular output inhibition in two patients (8%) and ventricular tracking in sixteen patients (64%). Pacemaker reprogramming abolished ventricular myopotential inhibition in all patients and stopped ventricular myopotential tracking in seven patients. We conclude that myopotentials can be analyzed and their effects ameliorated by a multiprogrammable pacemaker with electrogram telemetry capability.