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Reversion Mode Activation by Myopotential Sensing in a Ventricular Inhibited Demand Pacemaker
Author(s) -
ERKKILA KEVIN I.,
SINGH JANG B.
Publication year - 1985
Publication title -
pacing and clinical electrophysiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.686
H-Index - 101
eISSN - 1540-8159
pISSN - 0147-8389
DOI - 10.1111/j.1540-8159.1985.tb05722.x
Subject(s) - medicine , reversion , cardiology , ambulatory , holter monitor , electrocardiography , biochemistry , chemistry , gene , phenotype
Pacemaker follow‐up in a 72‐year‐old woman revealed occasional failure to sense and pace, which was confirmed by Holter monitor. Neither reprogramming the pacemaker sensitivity nor repositioning the lead resolved the problem. A recheck of the Holter recordings revealed pacing and sensing failures were concurrent with “baseline artifact,” suggestive of myopotentials. Furthermore, the inappropriate pacing spikes occurred at a rate of 90 pulses per minute (ppm). It was theorized that myopotential sensing was alternately inhibiting the pacer and activating the reversion mode, an asynchronous rate of 90 ppm. Reprogramming the unit to a lower sensitivity restored normal pacer function.