Premium
Increased plasmin‐α2‐antiplasmin levels indicate activation of the fibrinolytic system in systemic amyloidoses
Author(s) -
BOUMA B.,
MAAS C.,
HAZENBERG B. P. C.,
LOKHORST H. M.,
GEBBINK M. F. B. G.
Publication year - 2007
Publication title -
journal of thrombosis and haemostasis
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.947
H-Index - 178
eISSN - 1538-7836
pISSN - 1538-7933
DOI - 10.1111/j.1538-7836.2007.02457.x
Subject(s) - amyloidosis , medicine , fibrinogen , amyloid (mycology) , transthyretin , serum amyloid a protein , serum amyloid a , primary systemic amyloidosis , pathology , gastroenterology , immunology , systemic disease , inflammation , disease
*Laboratory for Thrombosis and Haemostasis, Department of Clinical Chemistry and Haematology, University Medical Center Utrecht and theInstitute for Biomembranes, Utrecht; Department of Rheumatology and Clinical Immunology, University Medical Center Groningen, Universityof Groningen, Groningen; and Department of Hematology, University Medical Center Utrecht, Utrecht, the NetherlandsTo cite this article: Bouma B, Maas C, Hazenberg BPC, Lokhorst HM, Gebbink MFBG. Increased plasmin-a2-antiplasmin levels indicate activationof the brinolytic system in systemic amyloidoses. J Thromb Haemost 2007; 5: 1139–42.See also Hammarstro¨m P. The bloody path of amyloids and prions. This issue, pp 1136–8.