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Characteristics of GABA B receptor binding sites on rat whole brain synaptic membranes
Author(s) -
Bowery N.G.,
Hill D.R.,
Hudson A.L.
Publication year - 1983
Publication title -
british journal of pharmacology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.432
H-Index - 211
eISSN - 1476-5381
pISSN - 0007-1188
DOI - 10.1111/j.1476-5381.1983.tb09380.x
Subject(s) - baclofen , muscimol , bicuculline , gabaa receptor , binding site , gabab receptor , chemistry , biophysics , gaba receptor , membrane , gamma aminobutyric acid , ligand (biochemistry) , biochemistry , receptor , agonist , biology
1 Saturable binding of (±)‐[ 3 H]‐baclofen and [ 3 H]‐γ‐aminobutyric acid ([ 3 H]‐GABA) to rat brain crude synaptic membranes has been examined by means of a centrifugation assay. 2 The binding of [ 3 H]‐baclofen could be detected in fresh or previously frozen tissue and was dependent on the presence of physiological concentrations of Ca 2+ or Mg 2+ although a lower affinity Na + ‐dependent component could also be observed. Both components probably reflect binding to receptor recognition sites. 3 The saturable portion of bound [ 3 H]‐baclofen formed 20.3 ± 6.9% of total bound ligand. This could be displaced by GABA (IC 50 =.0.04 μ m ), (−)‐baclofen (0.04 μ m ) and to a much lesser extent by (+)‐baclofen (33 μ m ). Isoguvacine, piperidine‐4‐sulphonic acid and bicuculline methobromide were inactive (up to 100 μ m ) and muscimol was only weakly active (IC 50 = 12.3 μ m ). 4 Saturable binding of [ 3 H]‐GABA increased on adding CaCl 2 or MgSO 4 (up to 2.5 m m and 5.0 m m respectively) to the Tris‐HCl incubation solution. This binding (GABA B site binding) was additional to the bicuculline‐sensitive binding of GABA (GABA A site binding) and could be completely displaced by (−)‐baclofen (IC 50 = 0.13 μ m ). 5 Increasing the Ca 2+ concentration (0 to 2.5 m m ) increased the binding capacity of the membranes without changing their affinity for the ligand. 6 The binding of [ 3 H]‐GABA to GABA B sites could be demonstrated in fresh as well as previously frozen membranes with a doubling of the affinity being produced by freezing. Further incubation with the non‐ionic detergent Triton‐X‐100 (0.05% v/v) reduced the binding capacity by 50%. 7 The pharmacological profile of displacers of [ 3 H]‐GABA from GABA B sites correlated well with that for [ 3 H]‐baclofen displacement. A correlation with data previously obtained in isolated preparations of rat atria and mouse vas deferens was also apparent. 8 It is concluded that [ 3 H]‐baclofen or [ 3 H]‐GABA are both ligands for the same bicuculline‐insensitive, divalent cation‐dependent binding sites in the rat brain.

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