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Idiopathic anetoderma with concomitant lesions of the Jadassohn‐Pellizzari and the Schweninger‐Buzzi types
Author(s) -
Haligah Charles E.K.,
Zouboulis Christos C.,
AbdelNaser Mohamed Badawy,
Detmar Michael,
Ehlers Günther,
Gollnick Harald
Publication year - 1994
Publication title -
journal of the european academy of dermatology and venereology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.655
H-Index - 107
eISSN - 1468-3083
pISSN - 0926-9959
DOI - 10.1111/j.1468-3083.1994.tb00382.x
Subject(s) - medicine , pathology , trunk , etiology , dermatology , ecology , biology
Anetoderma is a rare disease of unknown etiology, today classified as a subtype of the circumscribed elastotic disorders. The clinical picture shows atrophic patches located mainly on the upper trunk which characteristically give the palpating finger the sensation of a hernial orifice. Our patient with primary idiopathic anetoderma presented a rarely reported concomitance of the inflammatory type of lesions described by Jadassohn and Pellizzari together with the non‐inflammatory Schweninger‐Buxzi type. Historically and ultrastructurally, both kinds of lesions revealed similar pictures with a scanty perivascular lymphomononuclear cell infiltrate, with rarefied, partly fragmented elastic fibres and normal collagen fibrils. These findings indicate that these two types of lesions represent different stages of the same disease and not a simple association. Furthermore, they support the opinion that the classification of anetoderma into two separate Jadassohn‐Pellixzari and Schweninger‐Buzzi types may he unnecessary. After treatment with oral tetracycline 1 g/day for 3 weeks, no new lesions appeared; however, no improvement of the already present lesions occurred during a 6‐month follow‐up period.