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A BIOLOGICAL BASIS FOR RE‐STENOSIS AFTER PERCUTANEOUS TRANSLUMINAL ANGIOPLASTY: POSSIBLE UNDERLYING MECHANISMS
Author(s) -
Tennant M.,
McGeachie J. K.
Publication year - 1992
Publication title -
australian and new zealand journal of surgery
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.111
H-Index - 51
eISSN - 1445-2197
pISSN - 0004-8682
DOI - 10.1111/j.1445-2197.1992.tb00012.x
Subject(s) - medicine , angioplasty , percutaneous , stenosis , intimal hyperplasia , complication , balloon , radiology , cardiology , surgery , smooth muscle
Book reviews in this article: Intravascular catheterization is a commonly used diagnostic tool and percutaneous transluminal angioplasty is used to dilate stenosed blood vessels. Although these techniques are very successful diagnostically and therapeutically they may precipitate a number of acute and chronic complications. Chronic intimal changes following balloon angioplasty can result in re‐stenosis. Intimal hyperplasia can be a long‐term complication of both diagnostic and interventional vascular catheterization. This article details these long‐term structural changes, specifically relating experimental in vivo and in vitro changes to those seen clinically.