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The Haemodynamic Effects of Sodium Nitroprusside‐Induced Hypotension during Beta‐Adrenergic Blockade and Anaesthesia A Case Report
Author(s) -
Beierholm E. A.,
Sørensen M. Bredgaard,
Thorshauge C.
Publication year - 1978
Publication title -
acta anaesthesiologica scandinavica
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.738
H-Index - 107
eISSN - 1399-6576
pISSN - 0001-5172
DOI - 10.1111/j.1399-6576.1978.tb01373.x
Subject(s) - medicine , sodium nitroprusside , anesthesia , vascular resistance , propranolol , cardiac output , hemodynamics , pulmonary wedge pressure , stroke volume , blockade , blood pressure , heart rate , nitric oxide , receptor
We present the central haemodynamic data obtained during halothane/nitrous oxide anaesthesia in a patient under antihypertensive treatment with propranolol who underwent surgery for intracranial aneurysms in sodium nitroprusside‐induced hypotension. Cardiac output remained unchanged when hypotension was induced. Pulmonary capillary wedge pressure decreased. There was a slight increase in heart rate, as well as a minor decrease in stroke volume. Systemic vascular resistance decreased, but pulmonary vascular resistance remained unchanged. The response observed is discussed.