Premium
Biochemical studies on blossom end rot of tomatoes
Author(s) -
DEKOCK P. C.,
VAUGHAN D.,
HALL A.,
ORD B. G.
Publication year - 1980
Publication title -
physiologia plantarum
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.351
H-Index - 146
eISSN - 1399-3054
pISSN - 0031-9317
DOI - 10.1111/j.1399-3054.1980.tb03261.x
Subject(s) - peroxidase , hydroxyproline , catalase , cultivar , biology , horticulture , cell wall , chemistry , botany , enzyme , food science , biochemistry
The causes of cell wall necrosis in Blossom End Rot (BER) of tomatoes have been investigated by comparing enzyme activities in healthy and affected tissues in the cultivars Grenadier and Moneymaker. Changes in the mineral contents of the tomatoes were in accordance with those of calcium deficient tissues. The phenolic contents of the affected tissues were increased as also were caffeic and chlorogenic acids. Phenolase and peroxidase activities were higher in affected fruits white catalase activities were higher in healthy fruits. 14 C‐labelled leucine was incorporated into the proteins of the healthy fruits to the greatest extent but cell wall proteinbound hydroxyproline was greatest in affected fruits.