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The response of resistant and susceptible plants to diclofop‐methyl
Author(s) -
HOERAUF ROLAND A.,
SHIMABUKURO RICHARD H.
Publication year - 1979
Publication title -
weed research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.693
H-Index - 74
eISSN - 1365-3180
pISSN - 0043-1737
DOI - 10.1111/j.1365-3180.1979.tb01541.x
Subject(s) - chlorosis , avena fatua , shoot , avena , biology , meristem , phytotoxicity , hordeum vulgare , horticulture , agronomy , poaceae , botany , weed
Summary: Wild oat ( Avena fatua L. ) plants sprayed at the 2‐or 3‐leaf stages of growth with diclotop‐methyl developed chlorosis over the entire leaf blade of all leaves. The leaves became necfrotic 7 days after spraying Shool growth was inhibited. In wheat ( Triticum aesicum L cv.Waldron) discrete chlorotic areas developed only where the herbicide convicted the 2nd or 3rd leaf with no visible injury so new growth uf'ter treutment. Growth inhibition of susceptible oat ( Avena sativa L. cv. Garry) was sensitive to placement of diclutop‐methyl near the upica and meristematic sites of the plant. Chlorosis and necrosis were independent of herbicide placement. Selective herbicide placement induced chlorosis only or both chlorosis and growth inhibition Root growth in wild oat and barley ( Hordeum rulgare L. cv. Dickson) was strongly inhibited by 1–0 μM diclofop‐methyl. Wild oat shoots were killed when seedlings were root‐treated with 10 μM diclofop‐melhyl. The 100 μM rool treatment killed barley shoots but only stunted the growth of wheat shoots by approximately 50%. In root‐ireated wheat plants the shoots were turgid and developed a light purple colour, whereas in foliar‐treated plants the shoots developed discrete chlorotic areas.