Prolonged vitamin  D  intoxication: presentation, pathogenesis and progress | Zendy

Bell D. A. | Zendy; Crooke M. J. | Zendy; Hay N. | Zendy; Glendenning P. | Zendy

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Prolonged vitamin D intoxication: presentation, pathogenesis and progress

Author(s) -

Bell D. A.,

Crooke M. J.,

Hay N.,

Glendenning P.

Publication year - 2013

Publication title -

internal medicine journal

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.596

H-Index - 70

eISSN - 1445-5994

pISSN - 1444-0903

DOI - 10.1111/imj.12269

Subject(s) - medicine , hypercalcaemia , vitamin d and neurology , toxicity , parathyroid hormone , vitamin , pathogenesis , vitamin d deficiency , endocrinology , calcium

Vitamin D toxicity from unactivated vitamin D (calciferol) therapy is currently a rare cause of hypercalcaemia. However, the frequency of this event may increase as high‐dose unactivated vitamin D preparations become available. Prolonged vitamin D toxicity can cause reversible hypercalcaemia and partially reversible renal impairment. Parathyroid hormone may not be suppressed with unactivated vitamin D toxicity, especially if renal disease coexists.

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