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Relationship between Epicardial Adipose Tissue Thickness and Atrial Electromechanical Delay in Hypertensive Patients
Author(s) -
Akyel Ahmet,
Yayla Kadriye G.,
Erat Mehmet,
Sunman Hamza,
Doğan Mehmet,
Çimen Tolga,
Aytürk Mehmet,
Yeter Ekrem
Publication year - 2015
Publication title -
echocardiography
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.404
H-Index - 62
eISSN - 1540-8175
pISSN - 0742-2822
DOI - 10.1111/echo.12917
Subject(s) - medicine , epicardial adipose tissue , atrial fibrillation , cardiology , logistic regression , correlation , epicardial fat , adipose tissue , mathematics , geometry
Objectives Epicardial adipose tissue ( EAT ) and atrial electromechanical delay ( AEMD ) have been shown to be closely linked with atrial fibrillation. In this study, we aimed to investigate the relationship between EAT and AEMD . Methods Ninety‐six patients were included in this study. Echocardiographic measurements were performed, and laboratory parameters were recorded. Patients were divided into two groups according to their inter‐ AEMD values. Patients with an inter‐ AEMD value of <42.6 msec formed group 1 and patients with an inter‐ AEMD value of ≥42.6 msec formed group 2. Results The EAT thickness (3.7 ± 1.2 vs. 5.0 ± 1.6, P < 0.001) and LAVI (20.4 ± 2.9 vs. 24.5 ± 6.7, P = 0.001) were significantly higher in group 2. There was a significant positive correlation between AEMD parameters with EAT and LAVI . After multivariate logistic regression analysis, EAT ( OR : 1.505; 95% CI : 1.056–2.143, P = 0.023) and LAVI ( OR : 1.140; 95% CI : 1.018–1.277, P = 0.023) were found as independent predictors of prolonged AEMD . Conclusions We found that EAT thickness was closely related with AEMD , and we also found that EAT and LAVI were independent predictors of prolonged AEMD . These findings may be helpful to explain some pathogenic mechanisms in the development of AF .