Type I interferon regulates cytokine‐delayed neutrophil apoptosis, reactive oxygen species production and chemokine expression | Zendy

GlenAlty L. | Zendy; Moots R. J. | Zendy; Edwards S. W. | Zendy; Wright H. L. | Zendy

Open Access

Type I interferon regulates cytokine‐delayed neutrophil apoptosis, reactive oxygen species production and chemokine expression

Author(s) -

GlenAlty L.,

Moots R. J.,

Edwards S. W.,

Wright H. L.

Publication year - 2021

Publication title -

clinical & experimental immunology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.329

H-Index - 135

eISSN - 1365-2249

pISSN - 0009-9104

DOI - 10.1111/cei.13525

Subject(s) - chemokine , cxcl10 , proinflammatory cytokine , immunology , cytokine , tumor necrosis factor alpha , ccl3 , cxcl2 , biology , p38 mitogen activated protein kinases , interferon , cxcl1 , mapk/erk pathway , cancer research , ccl2 , inflammation , signal transduction , microbiology and biotechnology , chemokine receptor

Type‐I interferon regulates cytokine‐delayed neutrophil apoptosis, reactive oxygen species production and chemokine expression. This is mediated in part via activation of p38 MAPK.

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