Premium
Alcohol Intake and Reduced Mortality in Patients with Traumatic Brain Injury
Author(s) -
Cho Jin Seong,
Shin Sang Do,
Lee Eui Jun,
Song Kyoung Jun,
Noh Hyun,
Kim Yu Jin,
Lee Seung Chul,
Park Ju Ok,
Kim Seong Chun,
Hwang Seungsik
Publication year - 2016
Publication title -
alcoholism: clinical and experimental research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.267
H-Index - 153
eISSN - 1530-0277
pISSN - 0145-6008
DOI - 10.1111/acer.13065
Subject(s) - medicine , traumatic brain injury , hazard ratio , confounding , alcohol intake , alcohol , mortality rate , poison control , confidence interval , injury prevention , demographics , demography , emergency medicine , psychiatry , biochemistry , chemistry , sociology
Background The purpose of our study was to determine whether alcohol intake influences short‐term mortality in patients with traumatic brain injury ( TBI ), using a comprehensive trauma database. Methods We collected data from 7 emergency departments ( ED s) between J une 1, 2008 and M ay 31, 2010, using the same data form. Cases were included if they met the following criteria: (i) older than 15 and (ii) injuries including TBI . Demographics and outcomes were compared between patients with and without alcohol intake. We present the risk of mortality using hazard ratios and 95% confidence intervals. Results A total of 76,596 trauma patients visited the ED s during the study period; 12,980 patients were older than 15 and had TBI . There were 4,009 (30.9%) patients in the alcohol‐intake group, of whom 3,306 (82.5%) patients were male, 1,450 (36.2%) patients were moved by ambulance, and 1,218 (30.4%) patients’ injuries were intentional. The most frequent injury mechanism was falling down with alcohol intake and blunt injury without alcohol intake. Mortality rate was 1.0% with alcohol intake and 2.0% without alcohol intake. After adjusting for all factors related to mortality, the hazard ratio of mortality was 0.72 in the alcohol‐intake group. Conclusions Mortality rate due to TBI in the alcohol‐intake group appears to be lower compared to that in the no‐alcohol‐intake group after adjusting for main confounding variables.