The Shoot Apical Meristem Regulatory Peptide CLV3 Does Not Activate Innate Immunity | Zendy

Cécile Segonzac | Zendy; Zachary L. Nimchuk | Zendy; Martina Beck | Zendy; Paul T. Tarr | Zendy; Silke Robatzek | Zendy; Elliot M. Meyerowitz | Zendy; Cyril Zipfel | Zendy

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The Shoot Apical Meristem Regulatory Peptide CLV3 Does Not Activate Innate Immunity

Author(s) -

Cécile Segonzac,

Zachary L. Nimchuk,

Martina Beck,

Paul T. Tarr,

Silke Robatzek,

Elliot M. Meyerowitz,

Cyril Zipfel

Publication year - 2012

Publication title -

the plant cell

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 5.324

H-Index - 341

eISSN - 1532-298X

pISSN - 1040-4651

DOI - 10.1105/tpc.111.091264

Subject(s) - meristem , biology , flagellin , arabidopsis , innate immune system , regulator , arabidopsis thaliana , microbiology and biotechnology , immunity , leucine rich repeat , receptor , immune system , shoot , kinase , botany , mutant , immunology , biochemistry , gene

The Arabidopsis thaliana leucine-rich repeat receptor kinase FLAGELLIN SENSING2 (FLS2) is required for the recognition of bacterial flagellin in innate immunity. Recently, FLS2 was proposed to act as a multispecific receptor recognizing unrelated exogenous and endogenous peptide ligands, including CLAVATA3 (CLV3), a key regulator of shoot meristem stem cell production. Here, we report experimental evidence demonstrating that FLS2 does not recognize CLV3 and that the shoot apical meristem is immune to bacteria independently of CLV3 perception.

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