Unique Cellular and Mitochondrial Defects Mediate FK506-Induced Islet β-Cell Dysfunction | Zendy

Nassir Rostambeigi | Zendy; Ian R. Lanza | Zendy; Petras P. Dzeja | Zendy; Michael Deeds | Zendy; Brian A. Irving | Zendy; Honey V. Reddi | Zendy; Pranathi Madde | Zendy; Song Zhang | Zendy; Yan W. Asmann | Zendy; Jarett M. Anderson | Zendy; Jill M. Schimke | Zendy; K. Sreekumaran Nair | Zendy; Norman L. Eberhardt | Zendy; Yogish C. Kudva | Zendy

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Unique Cellular and Mitochondrial Defects Mediate FK506-Induced Islet β-Cell Dysfunction

Author(s) -

Nassir Rostambeigi,

Ian R. Lanza,

Petras P. Dzeja,

Michael Deeds,

Brian A. Irving,

Honey V. Reddi,

Pranathi Madde,

Song Zhang,

Yan W. Asmann,

Jarett M. Anderson,

Jill M. Schimke,

K. Sreekumaran Nair,

Norman L. Eberhardt,

Yogish C. Kudva

Publication year - 2010

Publication title -

transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.45

H-Index - 204

eISSN - 1534-6080

pISSN - 0041-1337

DOI - 10.1097/tp.0b013e3182094a33

Subject(s) - biology , mitochondrion , viability assay , microbiology and biotechnology , apoptosis , bioenergetics , microarray analysis techniques , programmed cell death , islet , medicine , endocrinology , insulin , gene expression , biochemistry , gene

To determine biological mechanisms involved in posttransplantation diabetes mellitus caused by the immunosuppressant tacrolimus (FK506).

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