Posttransplant Ischemia-Reperfusion Injury In Transplanted Heart Is Prevented By A Minibody to the Fifth Component of Complement | Zendy

Mariano Ferraresso | Zendy; Paolo Macor | Zendy; Marialuisa Valente | Zendy; Mila Della Barbera | Zendy; Fabio D’Amelio | Zendy; María Orietta Borghi | Zendy; Elena Raschi | Zendy; Paolo Durigutto | Zendy; Pier Luigi Meroni | Zendy; Francesco Tedesco | Zendy

Open Access

Posttransplant Ischemia-Reperfusion Injury In Transplanted Heart Is Prevented By A Minibody to the Fifth Component of Complement

Author(s) -

Mariano Ferraresso,

Paolo Macor,

Marialuisa Valente,

Mila Della Barbera,

Fabio D’Amelio,

María Orietta Borghi,

Elena Raschi,

Paolo Durigutto,

Pier Luigi Meroni,

Francesco Tedesco

Publication year - 2008

Publication title -

transplantation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.45

H-Index - 204

eISSN - 1534-6080

pISSN - 0041-1337

DOI - 10.1097/tp.0b013e31818a68e2

Subject(s) - complement system , medicine , necrosis , transplantation , ischemia , heart transplantation , allotransplantation , pathology , pharmacology , immunology , antibody

Complement activation has been implicated in the development of posttransplant ischemia-reperfusion (I/R) which is responsible for the delayed function of 20% to 30% of grafts. C5a and the terminal complement complex (TCC) are the complement activation products mainly involved in tissue injury caused by I/R.

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