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End-Tidal Carbon Dioxide Impacts Brain and Kidney Injury in Experimental Extracorporeal Cardiopulmonary Resuscitation (ECPR)
Author(s) -
Carl-Henrik Ölander,
Per Vikholm,
Petter Schiller,
Laila Hellgren
Publication year - 2020
Publication title -
shock
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.095
H-Index - 117
eISSN - 1540-0514
pISSN - 1073-2322
DOI - 10.1097/shk.0000000000001645
Subject(s) - medicine , extracorporeal cardiopulmonary resuscitation , cardiopulmonary resuscitation , resuscitation , extracorporeal membrane oxygenation , return of spontaneous circulation , anesthesia , cardiology , ventricular fibrillation
Extracorporeal membrane oxygenation-assisted cardiopulmonary resuscitation (ECPR) is proposed for cardiac resuscitation in selected cases. End-tidal carbon dioxide (ETCO2) is easily obtained during conventional cardiopulmonary resuscitation (CPR). We hypothesized that the level of ETCO2 during CPR would reflect the degree of brain and kidney damage following ECPR in experimental refractory cardiac arrest.

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