Fasudil Attenuates Myocardial Fibrosis in Association With Inhibition of Monocyte/Macrophage Infiltration in the Heart of DOCA/Salt Hypertensive Rats | Zendy

Kazuhiro Ishimaru | Zendy; Hitoshi Ueno | Zendy; Satoshi Kagitani | Zendy; Daisuke Takabayashi | Zendy; Masanobu Takata | Zendy; Hiroshi Inoue | Zendy

Open Access

Fasudil Attenuates Myocardial Fibrosis in Association With Inhibition of Monocyte/Macrophage Infiltration in the Heart of DOCA/Salt Hypertensive Rats

Author(s) -

Kazuhiro Ishimaru,

Hitoshi Ueno,

Satoshi Kagitani,

Daisuke Takabayashi,

Masanobu Takata,

Hiroshi Inoue

Publication year - 2007

Publication title -

journal of cardiovascular pharmacology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.762

H-Index - 100

eISSN - 1533-4023

pISSN - 0160-2446

DOI - 10.1097/fjc.0b013e318064f150

Subject(s) - fasudil , medicine , endocrinology , monocyte , fibrosis , hydroxyproline , myocardial fibrosis , infiltration (hvac) , chemistry , rho associated protein kinase , kinase , biochemistry , physics , thermodynamics

To determine the effects of fasudil, a Rho-kinase inhibitor, on mineralocorticoid-induced myocardial remodeling, we investigated whether fasudil would suppress myocardial fibrosis and inflammation in deoxycorticosterone-acetate (DOCA)/salt hypertensive rats.

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