Open AccessTrauma-Hemorrhagic Shock Mesenteric Lymph Induces Endothelial Apoptosis That Involves Both Caspase-Dependent and Caspase-Independent Mechanisms
Author(s) -
Marson T. Davidson,
Edwin A. Deitch,
Quan Lu,
György Haskó,
Billy Abungu,
Zoltán Németh,
Sergey B. Zaets,
Lawrence D. Gaspers,
Andrew P. Thomas,
Dakang Xu
Publication year - 2004
Publication title -
annals of surgery
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 4.153
H-Index - 309
eISSN - 1528-1140
pISSN - 0003-4932
DOI - 10.1097/01.sla.0000129341.94219.cf
Subject(s) - lymph , medicine , apoptosis , pathology , programmed cell death , cytotoxic t cell , lymph node stromal cell , caspase , endothelial stem cell , immunology , biology , biochemistry , in vitro
To determine the mechanism by which gut-derived factors present in mesenteric lymph from rats subjected to trauma-hemorrhagic shock (T/HS) induce endothelial cell death.