
THE MECHANISM OF INJURY IN A STEATOTIC LIVER GRAFT DURING COLD PRESERVATION1,2
Author(s) -
Tatsuya Fukumori,
Nobuhiro Ohkohchi,
Shigeki Tsukamoto,
Susumu Satoh
Publication year - 1999
Publication title -
transplantation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.45
H-Index - 204
eISSN - 1534-6080
pISSN - 0041-1337
DOI - 10.1097/00007890-199901270-00002
Subject(s) - phospholipid , steatosis , medicine , endocrinology , cholesterol , transplantation , cold storage , chemistry , polyunsaturated fatty acid , percoll , membrane fluidity , liver transplantation , viaspan , fatty acid , surgery , biochemistry , biology , membrane , centrifugation , horticulture
Fatty livers are more prone to primary nonfunction after transplantation. It is known that cell injury is strongly associated with alterations in the content and composition of membrane lipids. We assumed that plasma membrane (PM) fluidity, which is the most important property of the membrane, differed between fatty and normal livers.