Open AccessIL-13 IS OVEREXPRESSED IN EOSINOPHILIC ESOPHAGITIS AND INDUCES EOTAXIN-3 EXPRESSION BY A STAT6-DEPENDENT AND FLUTICASONE PROPIONATE-INHIBITED PATHWAY IN ESOPHAGEAL EPITHELIAL CELLS
Author(s) -
Carine Blanchard,
YiYing Wu,
Margaret H. Collins,
Philip E. Putnam,
Marc E. Rothenberg
Publication year - 2006
Publication title -
journal of pediatric gastroenterology and nutrition
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.206
H-Index - 131
eISSN - 1536-4801
pISSN - 0277-2116
DOI - 10.1097/00005176-200610000-00202
Subject(s) - eosinophilic esophagitis , eotaxin , interleukin 13 , fluticasone propionate , medicine , fluticasone , eosinophil , interleukin , cancer research , microbiology and biotechnology , immunology , pathology , cytokine , biology , asthma , disease