Angiotensin Receptor–Neprilysin Inhibition in Acute Myocardial Infarction
Author(s) -
Marc A. Pfeffer,
Brian Claggett,
Eldrin F. Lewis,
Christopher B. Granger,
Lars Køber,
Aldo P. Maggioni,
Douglas L. Mann,
John J.V. McMurray,
JeanLucien Rouleau,
Scott D. Solomon,
Philippe Gabríel Steg,
Otávio Berwanger,
Maja Čikeš,
Carmine G. De Pasquale,
Cara East,
Alberto Fernández,
Karola Jering,
Ulf Landmesser,
Roxana Mehran,
Béla Merkely,
Freny Vaghaiwalla Mody,
Mark C. Petrie,
Ivo Petrov,
Morten Schou,
Michele Senni,
David Sim,
Peter van der Meer,
Martin Lefkowitz,
Yig Zhou,
Jianjian Gong,
Eugene Braunwald
Publication year - 2021
Publication title -
new england journal of medicine
Language(s) - Uncategorized
Resource type - Journals
SCImago Journal Rank - 19.889
H-Index - 1030
eISSN - 1533-4406
pISSN - 0028-4793
DOI - 10.1056/nejmoa2104508
Subject(s) - neprilysin , myocardial infarction , valsartan , medicine , cardiology , heart failure , sacubitril , angiotensin converting enzyme , ace inhibitor , angiotensin receptor , angiotensin ii , receptor , enzyme , blood pressure , biochemistry , chemistry
In patients with symptomatic heart failure, sacubitril-valsartan has been found to reduce the risk of hospitalization and death from cardiovascular causes more effectively than an angiotensin-converting-enzyme inhibitor. Trials comparing the effects of these drugs in patients with acute myocardial infarction have been lacking.
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