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Mechanism of Repolarization Change During Initiation of Supraventricular Tachycardia
Author(s) -
LIN YENNJIANG,
TAI CHINGTAI,
CHIANG CHENEN,
LEE KUNTAI,
YUNIADI YOGA,
HUANG BIENHSIEN,
LIU TUYING,
LEE PICHIANG,
KUO JENYUAN,
CHEN SHIHANN
Publication year - 2004
Publication title -
journal of cardiovascular electrophysiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.193
H-Index - 138
eISSN - 1540-8167
pISSN - 1045-3873
DOI - 10.1046/j.1540-8167.2004.04049.x
Subject(s) - medicine , cardiology , supraventricular tachycardia , benign early repolarization , tachycardia , blood pressure , repolarization , electrocardiography , qrs complex , st segment , myocardial infarction , electrophysiology
Previous literature has documented the association between narrow QRS supraventricular tachycardia (SVT) and pronounced ST‐T segment change. The aim of this study was to evaluate repolarization changes during SVT initiation and demonstrate the possible mechanism. Methods and Results: Fifty‐one consecutive patients (20 men and 31 women; mean age 46.1 ± 16.4 years) with narrow QRS SVT (32 patients with AV nodal reentrant tachycardia and 19 patients with AV reentrant tachycardia) were included. We retrospectively analyzed the intracardiac recordings and ST‐T segment changes on 12‐lead surface ECGs during SVT initiation. Twenty‐six (51%) patients developed ST segment repolarization changes during SVT initiation. Patients with shorter baseline sinus cycle length, shorter tachycardia cycle length, elevated systolic blood pressure before tachycardia induction, and greater reduction of systolic blood pressure had a higher incidence of repolarization changes. However, multivariate analysis showed that reduction of systolic blood pressure after SVT induction was the only independent predictor of repolarization changes. Furthermore, the maximal degree of ST segment depression during SVT correlated with the reduction of systolic blood pressure (r = 0.75, P < 0.001). Conclusion: Repolarization changes during SVT initiation were caused mainly by concurrent hemodynamic change after SVT initiation with abrupt cycle length shortening.