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The Jk(a−b−) phenotype in New Zealand Polynesians
Author(s) -
Woodfield D. G.,
Douglas R.,
Smith J.,
Simpson A.,
Pinder L.,
Staveley J. M.
Publication year - 1982
Publication title -
transfusion
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.045
H-Index - 132
eISSN - 1537-2995
pISSN - 0041-1132
DOI - 10.1046/j.1537-2995.1982.22482251206.x
Subject(s) - polynesians , phenotype , allele , locus (genetics) , biology , genetics , medicine , gene , population , environmental health
The Kidd locus phenotype Jk(a‐b‐) was detected in 0.9 percent of Polynesians living in New Zealand. Over a period of 13 years, nine examples of anti‐Jk3 were detected, one of which caused a delayed hemolytic transfusion reaction. Other examples resulted in mild hemolytic disease of the newborn. The anti‐Jk3 reacted as an inseparable antibody, confirmed that inheritance of the Jk(a‐b‐) phenotype was best explained by the presence of a silent Jk allele.