Premium
P124S‐shaped kinking and constriction of the fetal ductus arteriosus with pericardial effusion
Author(s) -
Park M. Y.,
Kim Y. J.,
Woo B. H.,
Kim H. S.
Publication year - 2000
Publication title -
ultrasound in obstetrics and gynecology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.202
H-Index - 141
eISSN - 1469-0705
pISSN - 0960-7692
DOI - 10.1046/j.1469-0705.2000.00004-1-123.x
Subject(s) - medicine , ductus arteriosus , constriction , fetus , fetal echocardiography , pericardial effusion , cardiology , tricuspid insufficiency , tricuspid valve , pregnancy , prenatal diagnosis , genetics , biology
Usually fetal ductus arteriosus (DA) closure or constriction is caused by maternal medication of prostaglandin synthase inhibitor or corticosteroid. Although it is a rare condition, idiopathic DA constriction in the human fetus has been also reported. In our case, the patient was reffered at 26 weeks gestation due to pericardial effusion. The mother had not been received any medication of nonsteroidal anti‐inflammatory agents. The fetus had abnormally S‐shaped kinking and constriction of the DA, right arterial enlargement, right ventricular hypertrophy, tricuspid valve regurgitation and constrictive foramen ovale. An amniocentesis had been performed due to abnormal triple test and the fetal karyotype was 46XY. There was no evidence of fetal infection and other congenital malformation. Cesarean section was performed at 313 weeks gestation due to fetal hypoxia diagnosed by Doppler sonography and biophysical profile, and the baby was born in poor condition. On follow‐up, although the baby has right ventricular dysfunction for 2 month after birth, expected a good prognosis.