Premium
Urogenital infection and sperm motility
Author(s) -
Diemer T.,
Huwe P.,
Ludwig M.,
Hauck E. W.,
Weidner W.
Publication year - 2003
Publication title -
andrologia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.633
H-Index - 59
eISSN - 1439-0272
pISSN - 0303-4569
DOI - 10.1046/j.1439-0272.2003.00556.x
Subject(s) - motility , sperm motility , biology , genitourinary system , sperm , andrology , semen , fertility , immunology , medicine , microbiology and biotechnology , anatomy , genetics , population , environmental health
Summary. Male accessory sex gland infections are considered as potential hazards to male fertility. Various pathophysiological concepts have evolved from experimental and clinical studies that begin to explain the effects of bacteria and immunological events on the function of spermatozoa and sperm motility in particular. Besides direct influences of pathogenic bacteria on spermatozoa whose impact on the motility of human spermatozoa is reviewed herein, recent studies have identified and evaluated infectious mediators that appear to be responsible for specific molecular processes in infections that particularly affect the motility of spermatozoa. This review will focus in detail on direct bacterial effects of sperm motility, the role of seminal leucocytes and the impact of pro‐inflammatory cytokines on the motility of spermatozoa.