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Transient Suppression of Resting Corticosterone Levels Induces Sustained Increase of AVP Stores in Hypothalamic CRH‐Neurons of Rats
Author(s) -
Schmidt E. D.,
Janszen A. W. J W.,
Binnekade R.,
Tilders F. J. H.
Publication year - 1997
Publication title -
journal of neuroendocrinology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.062
H-Index - 116
eISSN - 1365-2826
pISSN - 0953-8194
DOI - 10.1046/j.1365-2826.1997.00618.x
Subject(s) - medicine , endocrinology , vasopressin , corticosterone , median eminence , stressor , corticotropin releasing hormone , neuropeptide , hypothalamus , neuroplasticity , biology , hormone , neuroscience , receptor
Previous studies showed that various stressors can induce delayed (days) and long‐lasting (weeks) increases of vasopressin (AVP) stores in terminals of CRH neurons in the external zone of the median eminence (ZEME) in adult rats. Here we tested whether this long‐lasting neuroplastic change can be induced by mechanisms other than stressor provoked transsynaptic activation of CRH neurons.