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Etiology of Trigeminal Neuralgia
Author(s) -
Toda Katsuhiro
Publication year - 2007
Publication title -
oral science international
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.256
H-Index - 13
eISSN - 1881-4204
pISSN - 1348-8643
DOI - 10.1016/s1348-8643(07)80007-3
Subject(s) - neurovascular bundle , trigeminal neuralgia , carbamazepine , medicine , etiology , anesthesia , surgery , dermatology , pathology , epilepsy , psychiatry
Neurovascular compression at the root entry zone accounts for more than 80% of trigeminal neuralgia (TN) cases, but not all patients with TN have neurovascular compression. Many non‐TN subjects have neurovascular contract at the root entry zone. TN is reported to occur in 0.9% to 4.5% of patients with multiple sclerosis (MS). In patients with TN, 1.7% to 15% of patients suffer from MS. The reported range for patients with TN due to tumors is from 0.8% to 11.6%. Because carbamazepine may relieve pain temporarily, relief of pain with carbamazepine does not exclude the diagnosis of a tumor or cyst. There are the peripheral cause theory, central cause theory, peripheral origin central pathogenesis theory, and multiple factors theory in the pathology of TN. Dental pain and/or treatment may trigger TM. Alveolar cavitational osteonecrosis may also cause TN.