Adenine Nucleotide Translocase 1 Deficiency Results in Dilated Cardiomyopathy With Defects in Myocardial Mechanics, Histopathological Alterations, and Activation of Apoptosis | Zendy

Nupoor Narula | Zendy; Michael V. Zaragoza | Zendy; Partho P. Sengupta | Zendy; Peng Li | Zendy; Nezam Haider | Zendy; Johan Verjans | Zendy; Katrina G. Waymire | Zendy; Mani A. Vannan | Zendy; Douglas C. Wallace | Zendy

Open Access

Adenine Nucleotide Translocase 1 Deficiency Results in Dilated Cardiomyopathy With Defects in Myocardial Mechanics, Histopathological Alterations, and Activation of Apoptosis

Author(s) -

Nupoor Narula,

Michael V. Zaragoza,

Partho P. Sengupta,

Peng Li,

Nezam Haider,

Johan Verjans,

Katrina G. Waymire,

Mani A. Vannan,

Douglas C. Wallace

Publication year - 2011

Publication title -

jacc. cardiovascular imaging

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 5.79

H-Index - 120

eISSN - 1936-878X

pISSN - 1876-7591

DOI - 10.1016/j.jcmg.2010.06.018

Subject(s) - medicine , dilated cardiomyopathy , adenine nucleotide translocator , endocrinology , cardiomyopathy , muscle hypertrophy , mitochondrion , cardiology , biology , heart failure , microbiology and biotechnology

the aim of this study was to test the hypothesis that chronic mitochondrial energy deficiency causes dilated cardiomyopathy, we characterized the hearts of age-matched young and old adenine nucleotide translocator (ANT)1 mutant and control mice.

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