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P1‐109: MITOCHONDRIAL DYSFUNCTION INDUCED BY A POSTTRANSLATIONALLY MODIFIED AMYLOID LINKED TO A FAMILIAL MUTATION IN AN ALTERNATIVE MODEL OF NEURODEGENERATION
Author(s) -
Todd Krysti,
Rostagno Agueda,
Ghiso Jorge
Publication year - 2014
Publication title -
alzheimer's and dementia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 6.713
H-Index - 118
eISSN - 1552-5279
pISSN - 1552-5260
DOI - 10.1016/j.jalz.2014.05.346
Subject(s) - neurodegeneration , amyloid (mycology) , mutation , amyloidosis , neurotoxicity , peptide , amino acid , amyloid precursor protein , p3 peptide , gene , biology , chemistry , biochemistry , microbiology and biotechnology , alzheimer's disease , medicine , toxicity , disease , pathology , botany , organic chemistry