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O2‐02–02: Soluble amyloid β‐protein dimers isolated directly from Alzheimer's disease patients potently impair synaptic plasticity and memory
Author(s) -
Shankar Ganesh M.,
Li Shaomin,
Mehta Tapan H.,
Garcia-Munoz Amaya,
Shepardson Nina E.,
Smith Imelda,
Brett Francesca M.,
Farrell Michael A.,
Rowan Michael J.,
Lemere Cynthia A.,
Regan Ciaran M.,
Walsh Dominic M.,
Sabatini Bernardo L.,
Selkoe Dennis J.
Publication year - 2008
Publication title -
alzheimer's and dementia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 6.713
H-Index - 118
eISSN - 1552-5279
pISSN - 1552-5260
DOI - 10.1016/j.jalz.2008.05.314
Subject(s) - long term potentiation , dendritic spine , synaptic plasticity , hippocampus , synapse , glutamate receptor , nmda receptor , chemistry , metabotropic glutamate receptor , alzheimer's disease , amyloid (mycology) , neurodegeneration , neuroscience , cerebral cortex , receptor , biochemistry , biology , medicine , disease , hippocampal formation , inorganic chemistry