Presence of fluorescent in situ hybridization abnormalities is associated with plasma cell burden in light chain amyloidosis | Zendy

Lindsay Hammons | Zendy; Ruta Brazauskas | Zendy; Marcelo C. Pasquini | Zendy; Mehdi Hamadani | Zendy; Parameswaran Hari | Zendy; Anita D’Souza | Zendy

Open Access

Presence of fluorescent in situ hybridization abnormalities is associated with plasma cell burden in light chain amyloidosis

Author(s) -

Lindsay Hammons,

Ruta Brazauskas,

Marcelo C. Pasquini,

Mehdi Hamadani,

Parameswaran Hari,

Anita D’Souza

Publication year - 2017

Publication title -

hematology/oncology and stem cell therapy

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.666

H-Index - 22

eISSN - 1658-3876

pISSN - 2589-0646

DOI - 10.1016/j.hemonc.2017.07.005

Subject(s) - amyloidosis , fluorescence in situ hybridization , medicine , al amyloidosis , amyloid (mycology) , pathology , multiple myeloma , fish <actinopterygii> , stage (stratigraphy) , plasma cell , bone marrow , gastroenterology , immunoglobulin light chain , biology , immunology , antibody , biochemistry , chromosome , gene , paleontology , fishery

To assess abnormalities found on CD138-enriched fluorescent in situ hybridization (FISH) studies on pre-treatment bone marrow in systemic amyloid light-chain amyloidosis (AL) and correlate findings between these abnormalities with organ involvement and 1-year survival.

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