z-logo
Premium
Phosphorylation of measles virus phosphoprotein at S86 and/or S151 downregulates viral transcriptional activity
Author(s) -
Sugai Akihiro,
Sato Hiroki,
Yoneda Misako,
Kai Chieko
Publication year - 2012
Publication title -
febs letters
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.593
H-Index - 257
eISSN - 1873-3468
pISSN - 0014-5793
DOI - 10.1016/j.febslet.2012.09.021
Subject(s) - phosphoprotein , phosphorylation , measles virus , transcription (linguistics) , nucleoprotein , viral protein , viral structural protein , biology , microbiology and biotechnology , virology , protein phosphorylation , subacute sclerosing panencephalitis , viral replication , rna polymerase ii , chemistry , protein kinase a , virus , viral entry , measles , gene expression , biochemistry , promoter , gene , linguistics , philosophy , vaccination
Measles virus phosphoprotein (P protein) is a cofactor of the viral RNA polymerase (L protein) that associates with the nucleoprotein–RNA complex to support viral transcription and replication. Here, we report a significant inverse correlation between the phosphorylation level of MV‐P protein and viral transcriptional activity. Upregulation of P protein phosphorylation resulted in reduction of viral transcription. Additionally, we found that strong phosphorylation at S86 and S151 of P protein, which may be generally prevented by association with nucleoprotein, downregulates the viral transcriptional activity. These findings suggest that P protein is involved in regulation of viral transcription through changes in its phosphorylation status.

This content is not available in your region!

Continue researching here.

Having issues? You can contact us here