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Defective relocalization of ALS2/alsin missense mutants to Rac1‐induced macropinosomes accounts for loss of their cellular function and leads to disturbed amphisome formation
Author(s) -
Otomo Asako,
Kunita Ryota,
Suzuki-Utsunomiya Kyoko,
Ikeda Joh-E,
Hadano Shinji
Publication year - 2011
Publication title -
febs letters
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.593
H-Index - 257
eISSN - 1873-3468
pISSN - 0014-5793
DOI - 10.1016/j.febslet.2011.01.045
Subject(s) - endosome , pinocytosis , microbiology and biotechnology , rac1 , mutant , retromer , autophagy , activator (genetics) , rab , biology , chemistry , endocytosis , gtpase , cell , biochemistry , gene , signal transduction , apoptosis , intracellular
ALS2 physically interacts with ALS2 by anti tag coimmunoprecipitation ( View interaction )
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