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BS69 negatively regulates the canonical NF‐κB activation induced by Epstein–Barr virus‐derived LMP1
Author(s) -
Ikeda Osamu,
Sekine Yuichi,
Mizushima Akihiro,
Oritani Kenji,
Yasui Teruhito,
Fujimuro Masahiro,
Muromoto Ryuta,
Nanbo Asuka,
Matsuda Tadashi
Publication year - 2009
Publication title -
febs letters
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.593
H-Index - 257
eISSN - 1873-3468
pISSN - 0014-5793
DOI - 10.1016/j.febslet.2009.04.022
Subject(s) - tradd , death domain , immunoprecipitation , traf2 , microbiology and biotechnology , signal transduction , tumor necrosis factor alpha , nf κb , kinase , epstein–barr virus , chemistry , biology , virus , virology , gene , apoptosis , tumor necrosis factor receptor , genetics , immunology , programmed cell death
MINT‐7032478: LMP1 (uniprotkb:P03230) physically interacts (MI:0218) with BRAM1 (uniprotkb:Q15326) by anti bait coimmunoprecipitation (MI:0006)

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