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Rotational Laxity Control by the Anterolateral Ligament and the Lateral Meniscus Is Dependent on Knee Flexion Angle: A Cadaveric Biomechanical Study
Author(s) -
Timothy Lording,
Gillian Corbo,
Dianne Bryant,
Timothy A. Burkhart,
Alan Getgood
Publication year - 2017
Publication title -
clinical orthopaedics and related research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.178
H-Index - 204
eISSN - 1528-1132
pISSN - 0009-921X
DOI - 10.1007/s11999-017-5364-z
Subject(s) - cadaveric spasm , medicine , tibia , anterolateral ligament , anterior cruciate ligament , femur , acl injury , orthodontics , anatomy , orthopedic surgery , cadaver , meniscus , biomechanics , knee joint , anterior cruciate ligament reconstruction , surgery , physics , incidence (geometry) , optics
Injury to the anterolateral ligament (ALL) has been reported to contribute to high-grade anterolateral laxity after anterior cruciate ligament (ACL) injury. Failure to address ALL injury has been suggested as a cause of persistent rotational laxity after ACL reconstruction. Lateral meniscus posterior root (LMPR) tears have also been shown to cause increased internal rotation of the knee.

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