GLP-2 Acutely Prevents Endotoxin-Related Increased Intestinal Paracellular Permeability in Rats | Zendy

Koji Maruta | Zendy; Takeshi Takajo | Zendy; Yasutada Akiba | Zendy; Hyder Said | Zendy; Emi Irie | Zendy; Ikuo Kato | Zendy; Atsukazu Kuwahara | Zendy; Jonathan D. Kaunitz | Zendy

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GLP-2 Acutely Prevents Endotoxin-Related Increased Intestinal Paracellular Permeability in Rats

Author(s) -

Koji Maruta,

Takeshi Takajo,

Yasutada Akiba,

Hyder Said,

Emi Irie,

Ikuo Kato,

Atsukazu Kuwahara,

Jonathan D. Kaunitz

Publication year - 2020

Publication title -

digestive diseases and sciences

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.14

H-Index - 124

eISSN - 1573-2568

pISSN - 0163-2116

DOI - 10.1007/s10620-020-06097-6

Subject(s) - paracellular transport , lipopolysaccharide , proinflammatory cytokine , intestinal permeability , chemistry , permeability (electromagnetism) , medicine , endocrinology , receptor , receptor antagonist , in vivo , endogeny , antagonist , pharmacology , inflammation , biochemistry , biology , microbiology and biotechnology , membrane

Circulating endotoxin (lipopolysaccharide, LPS) increases the gut paracellular permeability. We hypothesized that glucagon-like peptide-2 (GLP-2) acutely reduces LPS-related increased intestinal paracellular permeability by a mechanism unrelated to its intestinotrophic effect.

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