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An evaluation of a data mining signal for amyotrophic lateral sclerosis and statins detected in FDA's spontaneous adverse event reporting system
Author(s) -
Colman Eric,
Szarfman Ana,
Wyeth Jo,
Mosholder Andrew,
Jillapalli Devanand,
Levine Jonathan,
Avigan Mark
Publication year - 2008
Publication title -
pharmacoepidemiology and drug safety
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.023
H-Index - 96
eISSN - 1099-1557
pISSN - 1053-8569
DOI - 10.1002/pds.1643
Subject(s) - medicine , rosuvastatin , statin , atorvastatin , adverse event reporting system , adverse effect , placebo , simvastatin , fluvastatin , clinical trial , pravastatin , cerivastatin , context (archaeology) , retrospective cohort study , hmg coa reductase , pharmacoepidemiology , pharmacology , cholesterol , medical prescription , reductase , pathology , paleontology , biochemistry , chemistry , alternative medicine , biology , enzyme
Abstract Background We detected disproportionate reporting of amyotrophic lateral sclerosis (ALS) with HMG‐CoA‐reductase inhibitors (statins) in the Food and Drug Administration's (FDA) spontaneous adverse event (AE) reporting system (AERS). Purpose To describe the original ALS signal and to provide additional context for interpreting the signal by conducting retrospective analyses of data from long‐term, placebo‐controlled clinical trials of statins. Methods The ALS signal was detected using the multi‐item gamma Poisson shrinker (MGPS) algorithm. All AERS cases of ALS reported in association with use of a statin were individually reviewed by two FDA neurologists. Manufacturers of lovastatin, pravastatin, simvastatin, fluvastatin, atorvastatin, cerivastatin, and rosuvastatin were requested to provide the number of cases of ALS diagnosed during all of their placebo‐controlled statin trials that were at least 6 months in duration. Results There were 91 US and foreign reports of ALS with statins in AERS. The data mining signal scores for ALS and statins ranged from 8.5 to 1.6. Data were obtained from 41 statin clinical trials ranging in duration from 6 months to 5 years and representing approximately 200 000 patient–years of exposure to statin and approximately 200 000 patient–years of exposure to placebo. Nine cases of ALS were reported in statin‐treated patients and 10 cases in placebo‐treated patients. Conclusions Although we observed a data mining signal for ALS with statins in FDA's AERS, retrospective analyses of 41 statin clinical trials did not reveal an increased incidence of ALS in subjects treated with a statin compared with placebo. Copyright © 2008 John Wiley & Sons, Ltd.

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