z-logo
Premium
Clinical and pathological features in 15 Chinese patients with calpainopathy
Author(s) -
Luo SuShan,
Xi JianYing,
Lu JiaHong,
Zhao ChongBo,
Zhu WenHua,
Lin Jie,
Wang Yin,
Ren HuiMin,
Yin Bo,
Andoni Urtizberea J.
Publication year - 2011
Publication title -
muscle and nerve
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.025
H-Index - 145
eISSN - 1097-4598
pISSN - 0148-639X
DOI - 10.1002/mus.21908
Subject(s) - pathological , medicine , atrophy , pathology , muscle contracture , myopathy , muscle atrophy , muscle biopsy , muscle weakness , immunohistochemistry , weakness , calpain , dysferlin , biopsy , skeletal muscle , anatomy , biology , biochemistry , enzyme
Background: Calpainopathy is comprised of a group of myopathies caused by deficiency in calcium‐activated, neutral protease (calpain‐3). In this study we identify calpainopathy in a cohort of Chinese patients with unclassified myopathy and analyze its clinical and pathological features. Methods: Sixty‐six muscle biopsies were selected for combined Western blotting of dysferlin and calpain‐3 after immunohistochemical staining. Clinical and pathological parameters of 15 confirmed calpainopathy cases were determined. Results: The diagnosis of calpainopathy in 15 Chinese patients was confirmed by Western blot analysis. Fourteen subjects had progressive proximal muscle weakness; 1 presented with bilateral distal muscle atrophy of the lower extremities. Scapular winging was observed in 12 patients (80%), and joint contractures were found in 10 others (66.7%). Histopathological studies showed a high prevalence of lobulated fibers (66.7%). Conclusions: Chinese patients with calpainopathy share some common clinical and pathological features with the reported characteristics of non‐Chinese patients. Muscle Nerve, 2011

This content is not available in your region!

Continue researching here.

Having issues? You can contact us here